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New COVID-19 Jab Research Shows Spike Proteins In Heart Cells Within 48 Hours Of Inoculation

“New research out of Germany observing rat and human heart cells shows that within 48 hours of vaccination, the COVID-19 mRNA vaccines form spike proteins,” The Epoch Times reports.

“Spike proteins, made from the mRNA instructions inside the vaccines, were detected in the heart cells,” the outlet stated.

Cell abnormalities were found in the Pfizer and Moderna COVID-19 mRNA shots.

“The paper is a rapid communication paper, meaning it is a shorter scientific paper published more quickly than a standard research article. Scientists tend to use this format when they have findings that need to be shared immediately with the academic community,” Chief Nerd shared.

Via Chief Nerd:

“Within 48 hours, researchers detected spike proteins in both cell cultures and noticed abnormalities in heart contractions.

The researchers recorded heart contractions in a supplementary video, comparing normal contractions in an unvaccinated rat heart cell (1A) with vaccinated cells.

Pfizer-vaccinated cells displayed stronger, sustained contractions (1B) due to increased protein kinase A (PKA) activity.

The authors concluded that at the cellular level, the effects of the COVID-19 vaccines seemed to align closer with cardiomyopathy than with myocarditis. Cardiomyopathy is a condition where heart muscles become both structurally and functionally abnormal in the absence of other heart diseases. This differs from myocarditis and pericarditis, which occur when heart muscles become inflamed and damaged.”


“Pfizer-vaccinated cells displayed stronger, sustained contractions (1B) due to increased protein kinase A (PKA) activity. PKA levels are linked to heart performance; the higher the PKA level, the stronger the heart contractions,” The Epoch Times writes.

“Moderna-vaccinated cells developed irregular heart contractions and disrupted calcium regulation. The authors attributed the change in cell activity to disturbances in RyR2 receptors. These receptors play a key role in coordinating heart contractions using calcium,” the outlet added.

Per The Epoch Times:

While the paper indicated abnormality in mainly animal cells, the results strongly suggest vaccine cardiotoxicity.
“The present preclinical cardiac safety data point to the need for a reassessment of the risk–benefit ratio of RNA-based SARS-Cov-2 vaccines, given indication of their cardiotoxicity,” the authors wrote.

The heart abnormalities exhibited in the cells, likely caused by disruption to RyR2 and increased PKA protein levels, “are risk factors for sudden cardiac death, ventricular tachyarrhythmias, and contractile dysfunction,” they added.

“It’s very worrisome,” particularly since disruptions to the RyR2 receptor are directly linked to sudden cardiac death, Dr. McCullough said. “The pattern we’re seeing is people take the vaccine, and they die during exercise, or they die between 3 a.m. and 6 a.m.—again, where there’s a surge of catecholamines or stress in the body.”

Dr. McCullough added that the different pathways affected by the two vaccines suggest some underlying cell toxicity to the mRNA codes since Moderna and Pfizer vaccines have different mRNA codes, but both produce the same spike protein.

The peer-reviewed study, titled “Cardiac side effects of RNA-based SARS-CoV-2 vaccines: Hidden cardiotoxic effects of mRNA-1273 and BNT162b2 on ventricular myocyte function and structure,” states:

Background and Purpose

To protect against SARS-CoV-2 infection, the first mRNA-based vaccines, Spikevax (mRNA-1273, Moderna) and Comirnaty (BNT162b2, Pfizer/Biontech), were approved in 2020. The structure and assembly of the immunogen – in both cases, the SARS-CoV-2 spike (S) glycoprotein – are determined by a messenger RNA sequence that is translated by endogenous ribosomes. Cardiac side effects, which for the most part can be classified by their clinical symptoms as myo- and/or pericarditis, can be caused by both mRNA-1273 and BNT162b2.

Experimental Approach

As persuasive theories for the underlying pathomechanisms have yet to be developed, this study investigated the effect of mRNA-1273 and BNT162b2 on the function, structure, and viability of isolated adult rat cardiomyocytes over a 72 h period.

Key Results

In the first 24 h after application, both mRNA-1273 and BNT162b2 caused neither functional disturbances nor morphological abnormalities. After 48 h, expression of the encoded spike protein was detected in ventricular cardiomyocytes for both mRNAs. At this point in time, mRNA-1273 induced arrhythmic as well as completely irregular contractions associated with irregular as well as localized calcium transients, which provide indications of significant dysfunction of the cardiac ryanodine receptor (RyR2). In contrast, BNT162b2 increased cardiomyocyte contraction via significantly increased protein kinase A (PKA) activity at the cellular level.

Conclusions and Implications

Here we demonstrated for the first time, that in isolated cardiomyocytes, both mRNA-1273 and BNT162b2 induce specific dysfunctions that correlate pathophysiologically to cardiomyopathy. Both RyR2 impairment and sustained PKA activation may significantly increase the risk of acute cardiac events.


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